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UOO

DPhil in Cardiovascular Science (Secondary Prevention of Vascular Dysfunction and Disease)

✓ Fully Funded ⏰ Closing Soon 🎓 Bioengineering 🎓 Biomedical Sciences 🎓 Cardiovascular Science multi-omics cell biology regenerative medicine secondary prevention vascular disease ai imaging systems immunology cardiovascular research

Explore innovative cross-disciplinary methods to prevent and reverse vascular disease progression. Engage with cutting-edge multi-omics, AI imaging, and regenerative biology to identify novel therapeutic targets for cardiovascular health.

AI-generated overview

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Why This Research Matters

This research addresses critical unmet needs by targeting secondary prevention of vascular diseases, which are leading causes of death globally. Developing new therapeutic strategies based on multi-omics and AI technologies has the potential to significantly reduce cardiovascular risk and improve patient outcomes internationally.

Vascular Dysfunction Secondary Prevention Multi-Omics Imaging Systems Immunology Regenerative Approaches

Project Description

Project Overview

This multidisciplinary doctoral programme targets secondary prevention of vascular dysfunction and disease, such as coronary artery disease, stroke, and aortic aneurysm. Using transformative technologies—multi-omics and AI-enhanced imaging—it aims to discover new disease targets, mechanisms, and treatments to halt, stabilize, and reverse vascular disease progression.

What You Will Do

Students will undergo initial core research training and two laboratory rotations before engaging in a main research project across four interconnected themes: deep phenotyping and imaging, multi-omics and genetic discovery, systems immunology and therapeutic targeting, and vascular stability via cell biology and biomechanics. Collaboration is fostered across Oxford and Cambridge with symposia, entrepreneurship training, and industry interaction.

Expected Outcomes

Graduates will gain comprehensive cardiovascular science expertise with skills for academia, industry, biotechnology, healthcare, and policy. The programme aims to produce researchers capable of delivering innovative and translational impact on vascular disease prevention and treatment.

Why This Matters

Vascular diseases cause significant mortality and morbidity worldwide but have lacked major therapeutic progress in secondary prevention. This programme leverages cutting-edge technologies to develop urgently needed treatments, reducing cardiovascular risk and improving patient outcomes globally.

Entry Requirements

Applicants should hold or expect to obtain a first or upper second-class undergraduate degree (or equivalent) in Biomedical sciences, Bioengineering, Chemistry, Data science, Bioinformatics, or related life science subjects. Interdisciplinary interests and applicants from underrepresented groups in cardiovascular research are encouraged.

How to Apply

Applicants should submit CV, academic transcripts, a personal statement (up to 500 words) ranking top 3 project preferences, and three academic references. Applications to Oxford should be submitted via the programme website. Interviews will be held mid-late May 2026. Contact: Professor Nicola Smart at nicola.smart@dpag.ox.ac.uk.

Eligibility

UK/Home
EU
International

Supervisor Profile

PN
Professor Nicola Smart
University of Oxford
5314 Citations
36 h-index
Google Scholar

Professor Nicola Smart is a leading cardiovascular scientist at the University of Oxford specializing in vascular biology and disease mechanisms. Her research applies multidisciplinary methodologies including imaging, molecular biology, and genetics to understand and intervene in vascular dysfunction. She is recognized for advancing translational research in cardiovascular medicine.

Key Publications

2007 830 citations
Thymosin β4 induces adult epicardial progenitor mobilization and neovascularization
Demonstrated that Thymosin β4 promotes mobilization of adult epicardial progenitor cells and new blood vessel formation.
2011 826 citations
De novo cardiomyocytes from within the activated adult heart after injury
Provided evidence of new cardiomyocyte formation within the adult heart following injury.
2003 282 citations
Fraser syndrome and mouse blebbed phenotype caused by mutations in FRAS1/Fras1 encoding a putative extracellular matrix protein
Identified mutations in FRAS1 as the cause for Fraser syndrome and related phenotypes in mice.
2011 245 citations
Resident cardiac progenitor cells: at the heart of regeneration
Reviewed the role of resident cardiac progenitor cells in heart regeneration.
2008 238 citations
The stem cell movement
Discussed the dynamics and implications of stem cell mobilization and therapy.

Research Contributions

Thymosin β4 is essential for adult epicardial progenitor cell mobilization and neovascularization.
This finding advances cardiac regenerative medicine by identifying molecular targets for heart repair therapies.
New cardiomyocytes can form de novo in the adult heart post-injury.
This challenges old views of the heart as a non-regenerative organ and opens avenues for cardiac recovery research.
Mutations in extracellular matrix protein FRAS1 cause Fraser syndrome phenotypes.
Improved understanding of genetic bases of congenital disorders enabling better diagnosis and interventions.

More PhDs with Professor Nicola Smart

DPhil in Cardiovascular Science (VascularRewind: Secondary Prevention of Vascular Dysfunction and Disease)
University of Oxford Professor Nicola Smart Deadline: 01 May 2026

Advance cardiovascular science by developing innovative strategies to prevent and reverse vascular disease. Leverage multidisciplinary training and state-of-the-art technologies in a collaborative Oxford-Cambridge docto…

This research tackles critical gaps in vascular disease treatment by identifying new therapeutic targets and interventions. Successful outc…

3000+ citations · h25
Cardiovascular Science Secondary Prevention Multi-omics Imaging

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