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Viral Codon Usage: Comparative Analysis of Host-Virus Interactions in Chikungunya Infection

✓ Fully Funded 🎓 Genetics 🎓 Genomics 🎓 Molecular Biology bioinformatics arbovirus chikungunya virus host-virus interaction transcriptomics translatomics virus evolution molecular genetics

Explore host-specific responses to arboviruses by comparing human and mosquito cell interactions with chikungunya virus. Identify molecular pathways driving pathogenic versus tolerant outcomes and contribute to breaking viral transmission cycles.

AI-generated overview

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Why This Research Matters

This research addresses key knowledge gaps in arbovirus-host dynamics, crucial for developing targeted interventions against viral diseases affecting millions globally. By revealing molecular differences in viral tolerance and pathogenicity, it supports innovative strategies to impede virus spread and improve public health outcomes.

virus

Project Description

Project Overview

This PhD explores how arboviruses such as dengue, Zika, and chikungunya (CHIKV) produce vastly different outcomes in vertebrate hosts and mosquito vectors. CHIKV causes severe disease in humans but remains non-pathogenic in mosquitoes. The project aims to decipher molecular mechanisms underpinning these contrasting outcomes through comparative transcriptomic and translatomic profiling of infected human versus mosquito cells.

What You Will Do

The candidate will perform gene expression and translation analyses during CHIKV infection to map host-virus interactions. They will identify conserved pathways and host-specific mechanisms, focusing on alternative splicing and translational changes that drive pathogenic versus tolerant responses. Interdisciplinary training includes placements at GIMM for transcriptomic software/statistical methods, CNRS for computational modeling, and UCD for comparative bat immune biology studies.

Expected Outcomes

The project will generate a comprehensive comparative map detailing CHIKV-induced gene expression, splicing, and translation alterations in human and mosquito cells. Findings will clarify why CHIKV is pathogenic in humans but not mosquitoes, revealing targets for species-specific interventions to disrupt viral transmission.

Why This Matters

Understanding host-specific virus responses is vital to controlling arboviruses that threaten global health. Insights into molecular bases of viral tolerance in mosquitoes may inspire novel approaches to curtail spread of diseases impacting over 80% of populations in risk areas.

Entry Requirements

Background in bioinformatics, virology, molecular biology, or related fields. Strong analytical and computational skills preferred, especially experience or interest in transcriptomic data analysis and modeling approaches in infection biology.

How to Apply

Apply here: EVOMG-DN

Eligibility

UK/Home
EU
International

Supervisor Profile

PJ
Prof Juana Díez
Centre for Genomic Regulation (CRG)
3751 Citations
36 h-index
Google Scholar

Prof Juana María Díez Antón is an expert in virus-host molecular interactions, with extensive experience in RNA virus biology and antiviral mechanisms. Her research focuses on viral replication, antigenic diversification, and host protein roles in viral infection. She has a strong publication record, contributing to fundamental understanding of virus evolution and persistence.

Key Publications

2015 271 citations
Antiviral drug discovery: broad-spectrum drugs from nature
This paper surveys broad-spectrum antiviral drugs derived from natural sources, highlighting new avenues for antiviral therapies.
1993 182 citations
New observations on antigenic diversification of RNA viruses. Antigenic variation is not dependent on immune selection
The study provided new insights into antigenic diversification mechanisms of RNA viruses that occur independently of immune selection pressures.
2009 179 citations
Translation and replication of hepatitis C virus genomic RNA depends on ancient cellular proteins that control mRNA fates
This paper identified key ancient cellular proteins involved in hepatitis C virus RNA translation and replication, advancing understanding of virus-host interactions.
1988 176 citations
Coevolution of cells and viruses in a persistent infection of foot-and-mouth disease virus in cell culture
The research described the coevolutionary dynamics between cells and foot-and-mouth disease virus during persistent infections in cell culture.
2000 175 citations
Identification and characterization of a host protein required for efficient template selection in viral RNA replication
This study discovered a host protein critical for template selection during viral RNA replication, enhancing understanding of virus replication mechanisms.

Research Contributions

Identified ancient cellular proteins that regulate hepatitis C virus RNA translation and replication.
This finding enhances potential antiviral target discovery and therapeutic development against hepatitis C.
Demonstrated that antigenic variation in RNA viruses can occur independently of immune selection.
This insight affects vaccine design and understanding of viral evolution and immune escape mechanisms.
Discovered host proteins involved in viral RNA replication template selection.
Provides new targets for antiviral strategies by interfering with viral replication processes.

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